BLACKSBURG, Va., June 25, 2014 - The incarceration rate has nearly quadrupled since the U.S. declared a war on drugs, researchers say. Along with that, racial disparities abound. Black incarceration rates are more than six times higher than those for white Americans, according to the U.S. Bureau of Justice Statistics.
To explain these growing racial disparities, researchers at Virginia Tech are using the same modeling techniques used for infectious disease outbreaks to take on the mass incarceration problem, as detailed in a recent article in the Journal of the Royal Society Interface. By treating incarceration as an infectious disease, they demonstrate that small but significant differences in prison sentences can lead to large differences in incarceration rates.
Incarceration can be ‘transmitted’ to others. For instance, incarceration can increase family members’ emotional and economic stress or expose family and friends to a network of criminals, and these factors can lead to criminal activity.
Alternatively, “official bias” leads police and the courts to pay more attention to the incarcerated person’s family and friends, thereby increasing the probability they will be caught, prosecuted and processed by the criminal justice system, researchers said.
“Regardless of the specific mechanisms involved,” said Kristian Lum, a former statistician at the Virginia Bioinformatics Institute now working for private industry, “the incarceration of one family member increases the likelihood of other family members and friends being incarcerated.”
Building on this insight, incarceration is treated like a disease in the model and the incarcerated are infectious to their social contacts — their family members and friends most likely affected by their incarceration.
“Criminologists have long recognized that social networks play an important role in criminal behavior, the control of criminal behavior, and the reentry of prisoners into society,” said James Hawdon, a professor of sociology in the College of Liberal Arts and Human Sciences. “We therefore thought we should test if networks also played a role in the incarceration epidemic. Our model suggests they do.”
Synthesizing publically available data from a variety of sources, the researchers generated a realistic multi-generational synthetic population, contact networks, sentence lengths, and transmission probabilities.
The researchers’ model is comparable to real-world incarceration rates, reproducing many facets of the incarceration epidemic in the United States.
Both the model and actual statistics show large discrepancies in incarceration rates between black and white Americans and, subsequently, the likelihood of becoming a repeat offender is high. Comparisons like these can be used to validate the assumption that incarceration is infectious.
“Research clearly shows that this epidemic has had devastating effects on individuals, families, and entire communities,” Lum said. “Since our model captures the emergent properties of the incarceration epidemic, we can use it to test policy options designed to reverse it.”
Harsher sentencing may actually result in higher levels of criminality. Examining the role of social influence is an important step in reducing the growing incarceration epidemic.
This study appeared online June 25 in the Journal of the Royal Society Interface. Authors include Lum, a data scientist at DataPad, Samarth Swarup, an applied computer scientist at the Virginia Bioinformatics Institute, Stephen Eubank, a professor and the deputy director of the Network Dynamics and Simulation Science Laboratory; and Hawdon, a professor and director of the Center for Peace Studies and Violence Prevention at Virginia Tech.
Research reported in this publication was supported by the National Institute Of General Medical Sciences of the National Institutes of Health under Award Number U01GM070694 and the Defense Threat Reduction Agency Grant HDTRA1-11-1-0016. The content of the information does not necessarily reflect the official views of the federal government.
A university-level Research Institute of Virginia Tech, the Virginia Bioinformatics Institute was established in 2000 with an emphasis on informatics of complex interacting systems scaling the microbiome to the entire globe. It helps solve challenges posed to human health, security, and sustainability. Headquartered at the Blacksburg campus, the institute occupies 154,600 square feet in research facilities, including state-of-the-art core laboratory and high-performance computing facilities, as well as research offices in the Virginia Tech Research Center in Arlington, Va.
Written by Maureen Lawrence-Kuether.